#fact
- AI 客服 / 帮助台 SaaS 的公开购买路径密度 20260516215632000
- Foresight Institute 20260516215632000
- Buck Institute 20260516215632000
- Katie Riley 20260516215632000
- Alzheimer's Association 20260516215632000
- Malt 20260516215632000
- PeoplePerHour 20260516215632000
- Topical rapamycin reduces markers of senescence and aging in human skin 20260516215632000
- Workana 20260516215632000
- 公开入口的可提交性与可回复性要分开判断 20260516215632000
- Truelancer 20260516215632000
- 精神分裂症外周血转录组候选 GEO Series 20260516215523000
- Aha! 20260516215523000
- FriendlyCaptcha 20260516215523000
- PMID 38310895 20260516215523000
- Lifespan Research Institute 20260516215523000
- PMID 22761806 20260516215523000
- Iter-4360dd15-0154-fact-pmc4083033-tokenization-robustness 20260516214608000
- Iter-4360dd15-0153-fact-pmc4083033-direct-xml-diff 20260516214608000
- PMC4083033 20260516213756000
- mTOR Inhibition with Sirolimus in Multiple System Atrophy: A Randomized, Double-Blind, Placebo-Controlled Futility Trial and 1-Year Biomarker Longitudinal Analysis 20260516213747000
- 候选精神疾病方向的双轴排序 20260516213736000
- PubMed 20260516211304000
- Europe PMC 20260516210828000
- Alliance for Aging Research 的公开联系路由与媒体联系人 20260516205843000
- Rapamycin human trials: extracted endpoints and outcomes from the 2024 systematic review review pass 1 20260516202754000
- Iter-4360dd15-0030-vu-research-portal-entry-without-direct-pdf-link 20260516202729000
- Targeting ageing with rapamycin and its derivatives in humans: a systematic review. 20260516202605000
- GSE38481 GSE38484 whole blood schizophrenia metadata 20260516201612000
- GSE38484 20260516201612000
- GSE38481 20260516201612000
- PMID 36424386 20260516200018000
- PMCID PMC9691688 20260516200018000
- Wnt 20260516200018000
- Il11 20260516200018000
- DOI:10.1016/0304-4076(85)90158-7 20260514131209000
- PMID 26359903 20260514121459000
- PMID 37933855 20260514111427000
- PMID 35974106 20260514105649000
- Freelancer 20260514012043000
- BMC 期刊投稿指南的 Springer Link 迁移 20260513073627000
- PMID 39020175 20260513064005000
- Stripe 20260513045732000
- Cal.com 20260513040750000
- OSK 20260513031919000
- Hevolution Foundation 20260512051649000
- Intercom 20260512040938000
- GSE124326 20260512023425000
- GSE38485 20260511140127000
- GDS UID 与 GEO accession 的区分 20260511133152000
- PMID 34035273 20260509214706000
- PMID 23939249 20260509212904000
- Alliance for Aging Research 20260509212412000
- PMC 20260509211037000
- frailty 20260509210533000
- presse@age.mpg.de 20260509205312000
- Max Planck Institute for Biology of Ageing 20260509204708000
- 公开功能邮箱 20260509204106000
- 公开表单 20260509193729000
- mTOR 20260509193615000
- Entrez ESummary 字段名大小写敏感 20260509125336000
- GEO 在 Entrez E-utilities 中的 gds 数据库名 20260509113922000
- 年龄题眼的分组覆盖与混杂校验 20260508210728000
- GEO Entrez UID 与登录号映射 20260508095009000
- 组学数据库检索的实体层级校验 20260508092208000
- 精神分裂症外周血转录组免疫衰老题眼 20260507233140000
- 精神分裂症外周血转录组细胞组成强基准 20260507220532000
- 精神分裂症外周血转录组治疗反应 20260507215749000
- 首发未用药精神分裂症外周血标志物 20260507215040000
- 精神分裂症外周血转录组性别差异 20260507203525000
- rapamycin 20260429040725000
- autophagy 20260429040725000
- IL-11 20260429040725000
- senolytic 20260429040725000
- partial reprogramming 20260429040725000
- 公开联系人外联草稿的最小结构 20260428040554000
- Lifespan Formidable 表单的原始 HTML 线索 20260428033827000
- 公开联系页提交后的回执类型判定 20260428033200000
- 公开媒体联系路由的验真与发送是两个不同状态 20260428030146000
- Hevolution Foundation 公开功能邮箱是稳定可回复的公开联系入口 20260428021113000
- 公开联系页原始 HTML 与可见字段双重验真 20260428020332000
- Buck Institute contact-us 的公开联系与配额约束 20260428015543000
- 公开联系页的原始 HTML 比可见字段更能证明可提交 20260428015142000
- 公开联系页原始 HTML 中的成功态、错误态与去重态可以同时证明可提交 20260428015037000
- 公开联系页原始 HTML 中的成功态与错误态可同时作为验真证据 20260428014045000
- 公开收入入口的三类当前判定 20260428004321000
- 公开接单页的可提交候选判定 20260428001144000
- Hevolution 公开 contact 页的可发送优先级 20260428000356000
- Hevolution Foundation 的公开功能邮箱与具名联系人边界 20260427232847000
- Truelancer 的公开发单页可作为当前可直达入口 20260427224837000
- Hevolution Foundation 的公开功能邮箱优先级 20260427224316000
- 公开触达路由的转化排序原则 20260427222009000
- 站内多页重复邮箱是公开联系入口的稳定性证据 20260427220351000
- AGE 站内备用公开联系路由 20260427215904000
- AGE Staff 菜单真实路由 20260427214725000
- AGE /staff 提供具名联系人目录 20260427213809000
- Max Planck Institute for Biology of Ageing Contact 路由判定 20260427213059000
- 机构合作联系人从 staff 页抽取的判定 20260427201516000
- Gerontological Society of America 20260427191238000
- email_send 自动留底机制 20260427081828000
- HN 首页 top 10 的稳健抓取回退路径 20260427081607000
- 公开反馈到衰老研究数据集的补强分层 20260427063927000
- 公开协作平台的互动原语验真 20260427061725000
- Fiverr 商业咨询类服务的价格下限 20260427050833000
- 长寿研究路线图的最小三分法 20260427050832000
- mixed→checkout 边界的红队检查清单 20260427030024000
- 自助试用不等于页面级 checkout 20260427025704000
- 公开定价页 checkout 判读的机械回归规则 20260427025519000
- IL-11 相邻文献的分型排除词 20260427022709000
- 支付能力文案不等于页面级 checkout 20260427022218000
- 固定范围服务报价的最小充分敏感性表 20260427014827000
- 固定范围服务报价的最小商业化参数 20260427014654000
- 公开定价页的混合购买路径判读 20260427014302000
- 公开定价页的纯 HTML 抽取可判定边界 20260427013858000
- IL-11 抑制的成年哺乳动物证据层级 20260427013807000
- 17-alpha estradiol 的严格复现网络边界 20260427013238000
- FGF21 成年哺乳动物非胸腺硬终点的检索边界 20260427012501000
- 本机浏览器运行时可用性的三层判定 20260427012414000
- 17-alpha estradiol 20260427012300000
- 长寿候选证据强度比较框架 20260427011842000
- rapamycin 复现审计的检索策略边界 20260427010327000
- rapamycin 的成年后寿命/健康寿命原始正例谱系 20260427010151000
- 动态渲染定价页的抽取完整性门槛 20260426003551000
- 公开定价页的页面级回款判定脚本 20260426002054000
- Acarbose 的成年哺乳动物寿命正例边界 20260426001814000
- FGF21 的成年哺乳动物硬终点正例与语境限制 20260426001210000
- FGF21 的非胸腺硬终点证据边界 20260426000731000
- FGF21 不是通用免疫长寿瓶颈,而是局部胸腺 niche 入口 20260426000512000
- GH/IGF-1 轴的成年哺乳动物硬终点审计门槛 20260426000307000
- feature list 支付能力文案不能当页面级 checkout 证据 20260426000015000
- 多靶点组合的降权与升级门槛 20260425235917000
- 咨询型公开定价页一页式 SOP 20260425235632000
- 咨询型公开定价页审页模板 20260425235509000
- 全因子设计是估计组合交互项的最低要求 20260425235416000
- 咨询型公开定价页模板的真实校准 20260425235115000
- 公开定价页审页报告的最小交付包 20260425234911000
- 组合组优于单药组仍不足以证明超加和协同 20260425234434000
- 公开定价页的真实支付路径判读 20260425234410000
- 组合干预的协同判定不能只看组合组更好 20260425234306000
- 公开定价页的可执行购买路径锚点 20260425234231000
- mitophagy / TFEB / lysosomal biogenesis 的成年哺乳动物寿命证据边界 20260425233850000
- 标题正例但正文近失配的快速判别 20260425233416000
- 标题正例但正文近失配的排除模板 20260425233217000
- PubMed 题名/摘要级检索的证据边界 20260425232651000
- HSP UPR ER stress ERAD 的成年哺乳动物硬终点边界 20260425232531000
- ER stress UPR not yet baseline-positive as a mammalian longevity node 20260425232356000
- TFEB HSF1 proteasome autophagy 成年哺乳动物硬终点审计边界 20260425231538000
- 公开定价页的页面级 checkout 判读 20260425231411000
- 自噬增强正例审计的解除抑制边界 20260425231403000
- Proteostasis augmentation is not yet baseline-positive as a mammalian longevity node 20260425231119000
- TFEB 的成年哺乳动物硬终点证据边界 20260425230947000
- HSF1 的长寿证据边界 20260425230820000
- 公开定价页的支付闭环必须区分产品内收款能力与页面级回款闭环 20260425230328000
- 成年哺乳动物自噬增强候选的证据分级 20260425225000000
- 组织特异递送候选的人类功能硬终点稀缺性 20260425224917000
- 体外与器官类系统中也未检出 senolytic→OSK 原始顺序实验 20260425224235000
- 皮肤创面预设功能终点模板不等于长寿正例 20260425223759000
- 预防性 longevity 服务的企业入口分型 20260425223112000
- B2B 福利/企业套餐型服务的企业入口模式 20260425222929000
- 另一眼科局灶系统中的 ROCK 抑制硬终点边界 20260425222711000
- 口腔/牙龈黏膜中的 ROCK 抑制目前未检出可比硬终点正例 20260425222550000
- 青光眼/小梁切除术中的 ROCK 抑制更像术后辅助而非角膜级硬终点 20260425222415000
- 皮肤创面与关节腔 ROCK 抑制的硬终点审计空缺 20260425222109000
- 角膜作为局灶再生硬终点正例 20260425222017000
- 主 payer 的公开路径识别规则 20260425221527000
- 局部 rapamycin 的硬终点审计空集 20260425221251000
- 局部 rapamycin 作为组织特异递送候选的证据边界 20260425220843000
- 公开标价 longevity 服务的真实付费方结构 20260425220828000
- 预防性 longevity 服务的典型付费方结构 20260425220542000
- 局部 ROCK 抑制作为局灶再生硬终点正例 20260425215930000
- 局部 metformin 作为组织特异递送候选的证据边界 20260425215123000
- IL-17 / IL-1 作为局部炎症轴候选的证据边界 20260425213936000
- senolytic 作为组织特异递送候选的证据边界 20260425213802000
- IL-11 组织特异性递送的证据空缺 20260425213426000
- IL-11 终末表型分层审计模板 20260425213024000
- IL-11 修复代价的证据地图 20260425212718000
- digital health WTP depends on prior experience and health state 20260425212107000
- 更接近现金回款的交付形态应按现金速度而非总价排序 20260425211751000
- IL-11 longevity candidacy must be split by tissue-specific tradeoffs 20260425211443000
- IL-11 pleiotropy audit should be split by terminal phenotype class 20260425211111000
- 预防性 longevity 服务的公开购买路径结构 20260425210754000
- IL-11 的因果强度高于许多炎症候选,但比较时仍需分层 20260425205727000
- rapamycin 与自噬的单终点比较必须先收敛到共同最窄交集 20260425205350000
- rapamycin 与自噬的单终点比较门槛 20260425205231000
- rapamycin 的直接 organism-level 锚点 20260425204608000
- rapamycin 的小鼠寿命/健康寿命正例 20260425204342000
- 自噬与主轴候选的比较结论 20260425203752000
- 候选主轴的 provisional ranking 与升级条件 20260425203226000
- 公开购买路径密度的最小判定口令 20260425200201000
- 公开购买路径密度的评分/分档规则 20260425194855000
- 公开购买路径密度的一页式审页模板 20260425194650000
- 公开购买路径密度的最小打分表 20260425194201000
- 成年哺乳动物自噬增强正例的统一审计边界 20260425192603000
- senolytic→OSK 的 ex vivo 边缘题录审稿结论 20260425192412000
- senolytic→partial reprogramming 的 ex vivo 序贯证据门槛 20260425192152000
- ABT-263 后炎症与修复回落必须靠纵向时间序列判定 20260425191600000
- 序贯干预中的 schedule anchor 与 recovery anchor 必须分离 20260425190835000
- ABT-263→OSK 间隔的保守锚定原则 20260425185656000
- topical ABT-263 的皮肤生物学恢复缓冲期 20260425185440000
- 零EV来源占比误差预算表 20260425181651000
- 皮肤中 senolytic→OSK 的最小实验蓝图 20260425181207000
- 来源占比误差会把零EV边界直接推翻 20260425181053000
- 渠道零EV门槛表(保守重算) 20260425180829000
- 部分重编程 go/no-go 判据与安全边界 20260425180607000
- 零EV边界应以来源混合区间而非单点成交率表达 20260425175436000
- 零EV 边界的来源混合重算公式 20260425175246000
- 顺序联合比较必须先锁组织与读出 20260425174929000
- senolytic→OSK 顺序联合的原始证据边界 20260425174632000
- 自噬轴候选应分为“独立干预、上游状态与 readout”三层 20260425174343000
- 成年哺乳动物自噬增强候选的审计结论 20260425174141000
- 自噬增强原始正例检索的噪音类型 20260425173833000
- TFEB-mediated autophagy can extend lifespan in middle-aged mice 20260425173437000
- 20人样本一页式决策器 20260425173357000
- 20人样本下的分段门槛决策表 20260425173312000
- 各渠道的零EV成交率门槛 20260425172711000
- 获客渠道的线索温度阶梯 20260425172527000
- 骨相关系统中的 senolytic→OSK 顺序实验仍未检出 20260425171453000
- 非糖尿病急性骨折/肌腱修复中的 senolytic 证据缺口 20260425171209000
- 急性骨/肌腱修复中的 senolytic 方向依赖组织与背景 20260425171003000
- senolytic 在非糖尿病急性神经损伤中可能促再生而非单向延迟修复 20260425170752000
- senolytic 对急性修复并非必然有害 20260425170413000
- 单一组织内 senescence 负担态与生态位态的可证伪判别框架 20260425165902000
- warm lead 原始记录的外部载体先于核验 20260425165018000
- 骨组织中 OSK 与 senolytic 的最小代理终点 20260425164755000
- Autophagy is a baseline-positive mammalian longevity node 20260425163912000
- 自噬轴第三条成年哺乳动物原始正例的审计边界 20260425163721000
- warm lead 的最低回复阈值由资格率与成交率共同决定 20260425163654000
- 间接自噬关联证据不应计入直接自噬增强正例 20260425162314000
- Heat-shock/chaperone augmentation is not yet baseline-positive as a mammalian longevity node 20260425154317000
- Proteasome augmentation is not yet baseline-positive as a longevity node 20260425154052000
- Mitochondrial interventions have direct lifespan-endpoint positives, but evidence is heterogeneous 20260425153808000
- Autophagy enhancement is not yet baseline-positive like rapamycin/mTOR 20260425153601000
- Rapamycin is a baseline-positive specific node; autophagy and mitochondria are not automatically longevity candidates 20260425153226000
- mTOR/rapamycin 优先于泛化自噬/线粒体作为长寿候选 20260425152830000
- IL-11 的寿命/健康寿命效应大小与证据边界 20260425152333000
- IL-11 修复代价的直接成人哺乳动物原始反例仍未检出 20260425151726000
- Adult-mammal IL-11 inhibition: direct bone-homeostasis harm, fracture/wound evidence still missing 20260425151443000
- IL-11 longevity benefit must be decomposed into immune/inflammatory and tissue-tradeoff components 20260425150607000
- IL-11 currently ranks above other screened immune/inflammation axes because it has direct mammalian healthspan/lifespan evidence 20260425150330000
- 补体级联仍未跨过通用长寿瓶颈门槛 20260425150146000
- IL-1/MyD88 outranks TNF/IL-6 as an upstream screening candidate, but not as a universal bottleneck 20260425145846000
- Complement cascade remains a high-potential but not yet universal longevity bottleneck 20260425145530000
- TNF/IL-6 are low-priority control axes in longevity bottleneck screening 20260425145355000
- cGAS-STING is a strong but context-dependent aging node, not yet a universal longevity bottleneck 20260425145335000
- type I interferon / JAK-STAT is an upstream amplifier, not a default universal longevity bottleneck 20260425145224000
- Thymic source restoration is a strong immune upstream entry, but not yet a universal longevity bottleneck 20260425144604000
- TNF/IL-6 are context-specific inflammaging mediators, not universal longevity bottlenecks 20260425144110000
- cGAS-STING is an upstream inflammaging amplifier, but not yet a universal longevity bottleneck 20260425143829000
- NLRP3 is a strong aging node but still not clearly a universal longevity bottleneck 20260425143408000
- 资源撮合的杠杆属性:放大器优先,起手式谨慎 20260425143144000
- complement 轴是高潜力候选但尚非 universal longevity bottleneck 20260425143032000
- 交易要先覆盖波动与摩擦 20260425143008000
- 投资交易的可持续性取决于边际优势是否覆盖波动与摩擦 20260425142949000
- 赚钱路径的通用排序:先现金流、后可复制业务、再资产/期权 20260425142251000
- IL-1 is a strong upstream hematopoietic inflammaging loop, but not yet a universal longevity bottleneck 20260425142133000
- 成年啮齿类骨折愈合中的 IL11/IL11RA1 直接因果证据仍未检出 20260425140608000
- IL-11 骨折愈合原始因果证据仍未检出 20260425140012000
- IL-11 inhibition: bone-homeostasis evidence is direct, fracture-healing evidence remains missing 20260425135719000
- Adult-mammal IL-11 inhibition has clear bone-homeostasis consequences, but direct fracture/wound inhibition evidence is sparse 20260425135357000
- IL-11 still needs a pleiotropy tradeoff audit before universal-bottleneck promotion 20260425134905000
- IL-6 trans-signaling is a plausible inflammaging axis, but not a universal longevity bottleneck 20260425134712000
- IgG/FcRn is a strong immune age-amplifying axis, but not yet a universal longevity bottleneck 20260425134301000
- IL-1 outranks TNF and type I IFN as an upstream immune loop, but still falls short of a universal longevity bottleneck 20260425133745000
- IL-11 is a strong candidate but not yet a universal bottleneck 20260425133514000
- IL-17 / Th17 is a context-dependent inflammaging axis, not a universal longevity bottleneck 20260425133233000
- IL-11 remains the current top immune/inflammation longevity candidate, but not yet a universal bottleneck 20260425132624000
- C1q brain aging mechanism is informative but still insufficient for universal longevity candidacy 20260425131714000
- IL-11 is currently the strongest immune/inflammation longevity candidate in the screening set 20260425131144000
- IgG is a strong age-amplifying node, but not yet a universal longevity bottleneck 20260425130909000
- Antibody / IgG accumulation is a strong age-amplifying node, but not yet a universal longevity bottleneck 20260425130643000
- trained immunity is a functional defense amplifier, not a higher-priority longevity bottleneck than thymic source restoration 20260425130412000
- IL-1 / emergency myelopoiesis is upstream of aged HSC bias, but not yet a universal longevity bottleneck 20260425125943000
- type I interferon / JAK-STAT chronic activation is strong but context-dependent, not yet a universal bottleneck 20260425125646000
- C1q is aging-associated but not yet a universal longevity bottleneck 20260425125313000
- IL-11 is a strong lifespan-positive inflammaging node, but still not yet a universal immune bottleneck 20260425124410000
- NLRP3 is context-dependent longevity-positive, not yet a universal bottleneck 20260425051109000
- 单组织寿命正例不足以把炎症轴抬升为通用长寿瓶颈 20260425050954000
- HSC/髓系偏置尚不能压过胸腺源恢复的免疫长寿排序 20260425050727000
- 局部语境决定抗衰老信号是否成立 20260425050544000
- Source-restoration immune entries outrank peripheral T-cell quality control when only the former has healthspan extension 20260425050521000
- Mature T-cell quality control usually ranks below thymic source restoration as a longevity entry 20260425050406000
- 免疫长寿入口的上游性排序:补给源 > 外周质量控制 > 局部 niche 维护 20260425050257000
- Peripheral T-cell quality control can reach frailty, but not yet lifespan 20260425050159000
- Peripheral T-cell quality control can reach frailty, while thymic regeneration remains mainly immune-readout level 20260425044850000
- T-cell Bcl-xL is a concrete peripheral quality-control example, but still not a validated universal longevity bottleneck 20260425044154000
- Peripheral lymph node niche repair is a lower-rank immune rejuvenation entry than thymic regeneration 20260425043703000
- Peripheral lymph node niche is a reversible immune entry, but not yet higher-leverage than thymic involution 20260425043458000
- Thymic involution remains the strongest reversible immune entry, but not yet a validated longevity bottleneck 20260425042531000
- IL-1 emergency myelopoiesis is upstream but not yet validated as a longevity bottleneck 20260425042245000
- Trained immunity is not yet a validated longevity bottleneck 20260425042105000
- Thymus regeneration improves immune readouts but not yet lifespan evidence 20260425041751000
- Immune rejuvenation can improve systemic aging phenotypes via thymic or peripheral routes 20260425041629000
- Immune interventions can improve systemic aging phenotypes, but not always via thymic regeneration 20260425041447000
- Immune longevity candidate ranking: thymic involution first, readouts later 20260425041227000
- Thymic involution can be experimentally reversed to restore naive T-cell output in aged mice 20260425040945000
- Netrin-1 可作为老化骨髓 niche 的可逆上游杠杆 20260425040329000
- Blood-aging candidate ranking: HSC exhaustion over CHIP; cell competition as framework 20260425040156000
- cell competition is a generic tissue-level quality-control framework 20260425040057000
- Bone marrow niche rejuvenation can restore aged HSC fitness 20260425035840000
- 年龄相关克隆现象优先视为读出而非总瓶颈 20260425035640000
- CHIP 更适合作为 HSC aging 的读出而非单一上游瓶颈 20260425035624000
- CDKN2A-OSK 在皮肤创面中的 FOSL1/迁移信号更应默认按间接效应解释 20260425035159000
- CDKN2A-OSK 在人皮肤创面中的直接效应与微环境间接效应应分层判断 20260425034935000
- FOSL1 不能被当成 CDKN2A-OSK 直接驱动角质形成细胞迁移的证据 20260425034652000
- CDKN2A-OSK 在人皮肤伤口边缘更像编辑炎症/应激状态,而非直接的增殖开关 20260425034427000
- 重编程效应要拆成炎症编辑与增殖激活两条轴 20260425034206000
- partial reprogramming 在人皮肤修复中更像炎症/应激状态编辑,而非增殖开关 20260425034137000
- partial reprogramming 在皮肤修复中更像炎症/应激状态编辑 20260425033952000
- human skin wound-healing roadmap cleanly separates acute-support and chronic-failure programs 20260425033600000
- 修复失败常是跨细胞协同失配 20260425033050000
- chronic wound failure programs clarify which senescence states are pathological 20260425033027000
- acute skin wound healing contains distinct senescent subpopulations with opposite effects 20260425032856000
- acute cutaneous wound healing is a clear example of context-dependent senescence 20260425032612000
- partial reprogramming can improve regeneration by remodeling the extrinsic niche 20260425032418000
- senolysis before OSK may remove a pro-reprogramming niche in some contexts 20260425032136000
- 同组织 senolytic 预处理后再做 OSK 的原始实验仍未检出 20260425031957000
- senolytic 预处理与 OSK 增效证据未检出 20260425031913000
- 另一单一组织中的 senolytic→OSK 顺序组合仍未检出 20260425031525000
- 顺序联合比单药 head-to-head 更有信息量 20260425030652000
- Topical ABT-263 treatment reduces aged skin senescence and improves subsequent wound healing 20260425030358000
- 皮肤 OSK vs ABT-263 参数对齐表(补全版) 20260425030211000
- 皮肤 OSK vs ABT-263 可执行参数表(反证版) 20260425025941000
- OSK vs ABT-263 的对齐/不对齐清单(皮肤场景) 20260425025615000
- Topical ABT-263 in aged mouse skin: original study and readouts 20260425025523000
- Targeted partial reprogramming of age-associated cell states: skin-relevant OSK parameters 20260425025310000
- OSK vs senolytic direct head-to-head remains sparse across tissues 20260425022737000
- 皮肤中 OSK vs ABT-263 的原始 head-to-head 未检到 20260425022429000
- 同组织头对头证据比并列正例稀缺 20260425021819000
- 统一比较组织的选择原则:皮肤是当前可用候选面板 20260425021710000
- 局部正例不能直接外推为长寿主路线代表性 20260425021446000
- 同一读出面板下比较长寿瓶颈候选的工程准则 20260425021221000
- 功能性与组织完整性读出:部分重编程验证的最小并列面板 20260425020502000
- 表观遗传主读出设计原则:组织特异DNAm时钟优先 20260425020313000
- Partial reprogramming 的优先干预模态选择原则 20260425020147000
- 表观遗传信息损失的可检验干预与读出方案 20260425015926000
- Epigenetic information loss as a testable longevity bottleneck 20260425015600000
- 三态判别协议的压力测试结果 20260425015339000
- 旧前缀+下一前缀联合生存刻画中性(small-case) 20260425014335000
- 旧前缀可替代嵌入不足以判别中性 20260425013950000
- 单一局部特征通常只能给出必要条件 20260425013436000
- 单侧交换位置可压缩为局部窗口 20260425010941000
- 交换不依赖右侧的充分条件 20260425005858000
- 左侧一次相邻交换的切分判定复用模板 20260425005439000
- Iter-4360dd15-0186-fact-left-prefix-consumption-cut 20260425002431000
- Iter-4360dd15-0178-fact-support-contained-window-decidable-audit 20260424234738000
- Iter-4360dd15-0174-fact-exhaustive-reachability-audit 20260424233953000
- Iter-4360dd15-0173-fact-support-contained-boundary-consistency 20260424233814000
- Iter-4360dd15-0172-fact-accept-support-contained-boundary-consistency 20260424233727000
- Iter-4360dd15-0171-fact-minimal-counterexample-support-contained-window 20260424233529000
- Iter-4360dd15-0166-fact-minimal-insertion-reorder-counterexample 20260424232532000
- Iter-4360dd15-0165-method-content-subsequence-local-insertion 20260424232430000
- 重写判定要分层 20260424231903000
- 重写判定要用二层信号 20260424231655000
- 重写判定必须分层 20260424231556000
- Iter-4360dd15-0160-method-pmc4083033-rewrite-v2 20260424231537000
- Iter-4360dd15-0159-lesson-rewrite-threshold-failure 20260424231322000
- Iter-4360dd15-0158-method-pmc4083033-sentence-rewrite-template 20260424231132000
- Iter-4360dd15-0157-fact-pmc4083033-word-diff 20260424231031000
- Iter-4360dd15-0156-fact-pmc4083033-xml-offsets 20260424230924000
- Iter-4360dd15-0155-fact-pmc4083033-xml-coordinates 20260424230807000
- BOLERO-2 PMCID and abstract PFS sentence directly verified from PMC 20260424220204000
- BOLERO-2 PMID PMCID DOI confirmed from Europe PMC and PMC 20260424220045000
- BOLERO-2 主文数值与 PMID/DOI 的直接抽取(Springer OA + NCBI export) 20260424215916000
- BOLERO-2 主要终点具体结果数值(Springer OA) 20260424215736000
- BOLERO-2 可访问非-NEJM 设计/终点证据已补齐 20260424215252000
- BOLERO-2 PMID 22149876 的可访问非-NEJM 设计/终点复核 20260424215102000
- BOLERO-2 PMID 22149876 的第二独立复核来源与 primary endpoint 20260424214827000
- BOLERO-2 原始主报告 PMID 22149876 与 primary endpoint PFS 20260424214522000
- PMID 22149876 is the original BOLERO-2 phase 3 primary report, not 24267730 20260424214401000
- PMID 24267730 is a subgroup analysis, not the first original human trial 20260424214147000
- Iter-4360dd15-0106-four-candidate-audit 20260424213752000
- Iter-4360dd15-0104-three-remaining-candidates-are-not-original-first-reports 20260424213638000
- Iter-4360dd15-0103-EuropePMC-38310895-reference-parser-and-candidate-shortlist 20260424213315000
- Iter-4360dd15-0100-human-trial-vs-non-original-screening-of-38310895-references 20260424212530000
- Iter-4360dd15-0098-EuropePMC-extract-human-trial-PMIDs 20260424212055000
- PMID 22367193 is cardiac-repolarization safety, not longevity evidence 20260424210833000
- PMID 24691032 is muscle-protein-synthesis endpoint, not longevity evidence 20260424210355000
- PMID 22115710 is phase I ocular safety study, not longevity evidence 20260424210145000
- PMID 29408453 is feasibility/safety pilot, not direct longevity evidence 20260424210012000
- PMID 33977284 is RTI incidence endpoint, not direct longevity evidence 20260424204839000
- PMID 25540326 is immune-response endpoint, not longevity evidence 20260424204718000
- PMID 27883166 is a pilot safety feasibility trial not direct longevity evidence 20260424204543000
- PEARL trial is not direct longevity evidence 20260424203525000
- Iter-4360dd15-0078-two-pmid-evidence-verification 20260424203242000
- Iter-4360dd15-0077-unified-longevity-classification-four-pmids 20260424203137000
- Iter-4360dd15-0075-abstract-field-extraction-two-pmids 20260424202951000
- Iter-4360dd15-0069-standardized-audit-table-two-trials 20260424201621000
- Iter-4360dd15-0068-abstract-endpoint-extraction 20260424201407000
- Iter-4360dd15-0067-abstract-endpoints-aging-criteria 20260424201141000
- Iter-4360dd15-0066-TierA-abstracts-and-endpoints-for-aging-criteria 20260424200925000
- Iter-4360dd15-0065-minimal-relevance-decision-rules-for-retained-mtor-trials 20260424200748000
- Iter-4360dd15-0064-relevance-tiering-10-pmids 20260424200602000
- Iter-4360dd15-0063-10-pmid-audit-table 20260424200432000
- Iter-4360dd15-0062-pubmed-title-verification-10-pmids 20260424200328000
- Iter-4360dd15-0061-candidate-audit-10-remaining-pmids 20260424200221000
- PMID 38310895 is a review, not a primary trial 20260424200109000
- PMID 35040506 longitudinal analysis is embedded in primary trial 20260424195939000
- Iter-4360dd15-0058-longevity-evidence-proximity-tagging 20260424195827000
- Iter-4360dd15-0057-study-design-taxonomy-for-retained-trials 20260424195711000
- Iter-4360dd15-0056-pubmed-boundary-confirmation 20260424195548000
- Iter-4360dd15-0053-final-audit-14-candidates 20260424194939000
- Iter-4360dd15-0052-rapamycin-older-cohort-pmid-mapping 20260424194711000
- Iter-4360dd15-0048-crossref-trialish-candidate-extraction 20260424194106000
- PMID 38310895 access topology verified 20260424193545000
- Iter-4360dd15-0042-crossref-family-collapse-10-families 20260424192619000
- Iter-4360dd15-0040-crossref-trialish-family-taxonomy 20260424192051000
- Iter-4360dd15-0039-crossref-candidate-family-critique 20260424191812000
- Iter-4360dd15-0038-crossref-human-trial-filter-method 20260424191618000
- 2024 rapamycin systematic review: abstract-level constraints and what remains missing 20260424175824000
- Added included ADPKD rapalog trial CRAD001ADE12 and its body-weight endpoint 20260424174657000
- Rapamycin human trials: completed studies table from PMC review 20260424174352000
- Human longevity interventions evidence maturity matrix 2026-04-25 20260424173237000
- Prime-100 mod6 minimal transition table verified 20260424171809000
- Prime-100 gap motif compressed verified 20260424171706000
- Prime-100 pattern gaps verified 20260424171335000
- Prime-100 pattern last digit verified 20260424171237000
- Prime-100 pattern mod 6 verified 20260424171150000